We hypothesized that alterations in cellular protein S-nitrosylation may contribute to hyperglycemia-induced endothelial dysfunction.
我们假设细胞蛋白s -亚硝基化的变化会引起高血糖介导的内皮功能障碍。
One area is S-nitrosylation, where we've been doing some work with a group at Johns Hopkins, the protein, transglutaminase2, seems to be effected by this process S-nitrosylation.